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dc.contributor.advisor Wang, Xiao-Fan en_US
dc.contributor.author Yong, ST
dc.contributor.author Wang, XF
dc.coverage.spatial United States
dc.date.accessioned 2012-01-10T15:57:17Z
dc.date.issued 2012
dc.identifier http://www.ncbi.nlm.nih.gov/pubmed/22761665
dc.identifier PONE-D-11-22003
dc.identifier.citation PLoS One, 2012, 7 (6), pp. e38085 - ?
dc.identifier.uri http://hdl.handle.net/10161/4958
dc.description Dissertation en_US
dc.description.abstract Scythe/BAT3 is a member of the BAG protein family whose role in apoptosis has been extensively studied. However, since the developmental defects observed in Bat3-null mouse embryos cannot be explained solely by defects in apoptosis, we investigated whether BAT3 is also involved in cell-cycle progression.
dc.format.extent e38085 - ?
dc.language eng
dc.relation.ispartof PLoS One
dc.relation.isversionof 10.1371/journal.pone.0038085
dc.subject Apoptosis
dc.subject Blotting, Western
dc.subject Bone Neoplasms
dc.subject Cell Cycle
dc.subject Cell Proliferation
dc.subject Cyclin-Dependent Kinase Inhibitor p21
dc.subject DNA Replication
dc.subject Flow Cytometry
dc.subject Fluorescent Antibody Technique
dc.subject Humans
dc.subject Molecular Chaperones
dc.subject Osteosarcoma
dc.subject Phosphorylation
dc.subject RNA, Small Interfering
dc.subject Tumor Cells, Cultured
dc.title A novel, non-apoptotic role for Scythe/BAT3: a functional switch between the pro- and anti-proliferative roles of p21 during the cell cycle.
dc.type Journal Article
dc.department Molecular Cancer Biology en_US
duke.embargo.months 24 en_US
pubs.author-url http://www.ncbi.nlm.nih.gov/pubmed/22761665
pubs.issue 6
pubs.organisational-group /Duke
pubs.organisational-group /Duke/School of Medicine
pubs.organisational-group /Duke/School of Medicine/Basic Science Departments
pubs.organisational-group /Duke/School of Medicine/Basic Science Departments/Pharmacology & Cancer Biology
pubs.organisational-group /Duke/School of Medicine/Institutes and Centers
pubs.organisational-group /Duke/School of Medicine/Institutes and Centers/Duke Cancer Institute
pubs.volume 7
dc.identifier.eissn 1932-6203

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