Browsing by Author "Stefanovic, Alexandra"
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Item Open Access Post-transplant lymphoproliferative disorder presented as small bowel intussusception in adult liver transplant patient.(J Korean Surg Soc, 2012-01) Joo, Sun Hyung; Acun, Zeki; Stefanovic, Alexandra; Blieden, Clifford R; Ikpatt, Offiong F; Moon, JangIntestinal obstruction after liver transplant is a rare complication, with diverse clinical manifestations. Intestinal adhesion is the most common cause. However, internal hernia, abdominal wall hernia, and neoplasm are also reported. Intussusception is another rare cause of intestinal obstruction, which has been reported primarily in pediatric patients. Herein, we report a case of intestinal obstruction from intussusception in an adult liver transplant patient associated with post-transplant lymphoproliferative disorder.Item Open Access Radiation therapy for primary cutaneous γδ T-cell lymphoma: Case report and literature review.(JAAD case reports, 2019-07) Kelsey, Chris R; Wang, Endi; Stefanovic, Alexandra; Kheterpal, MeenalItem Open Access The potential role of complements in cocaine-induced thrombotic microangiopathy.(Clinical kidney journal, 2018-02) Dejman, Adriana; Alavi, Seyed Navid; Thomas, David B; Stefanovic, Alexandra; Asif, Arif; Nayer, AliThrombotic microangiopathy (TMA) is a rare disorder characterized by microvascular injury and occlusion resulting in tissue ischemia and dysfunction. TMA occurs in a variety of settings including cocaine use. Although cocaine is widely used in the United States, cocaine-associated TMA is only rarely reported. Therefore, other factors may predispose cocaine users to the development of TMA. Emerging evidence indicates that cocaine activates complements. Therefore, complement activation may contribute to the development of cocaine-induced TMA. Here, we report a cocaine user who presented with renal failure. Renal biopsy demonstrated TMA. Laboratory tests revealed reduced serum complement C3 and normal complement C4 levels indicative of alternative complement activation. We postulate that complement activation is involved in the pathogenesis of cocaine-induced TMA.