Browsing by Author "Martin, B"

Genomic deletion of GIT2 induces a premature age-related thymic dysfunction and systemic immune system disruption.

Siddiqui, S; Lustig, A; Carter, A; Sankar, M; Daimon, CM; Premont, Richard Thomas; Etienne, H; ... (18 authors) (Aging (Albany NY), 2017-03-04)

Recent research has proposed that GIT2 (G protein-coupled receptor kinase interacting protein 2) acts as an integrator of the aging process through regulation of 'neurometabolic' integrity. One of the commonly accepted hallmarks ...

GIT2 Acts as a Systems-Level Coordinator of Neurometabolic Activity and Pathophysiological Aging.

Becker, KG; Chadwick, W; Cong, WN; Daimon, CM; Janssens, J; Lehrmann, E; Martin, B; ... (14 authors) (Front Endocrinol (Lausanne), 2015)

Aging represents one of the most complicated and highly integrated somatic processes. Healthy aging is suggested to rely upon the coherent regulation of hormonal and neuronal communication between the central nervous system ...

Hippocampal Transcriptomic and Proteomic Alterations in the BTBR Mouse Model of Autism Spectrum Disorder.

Becker, KG; Crawley, JN; Daimon, CM; Jasien, Joan M; Martin, B; Maudsley, S; Silverman, JL; ... (9 authors) (Front Physiol, 2015)

Autism spectrum disorders (ASD) are complex heterogeneous neurodevelopmental disorders of an unclear etiology, and no cure currently exists. Prior studies have demonstrated that the black and tan, brachyury (BTBR) T+ Itpr3tf/J ...

The effects of aging on the BTBR mouse model of autism spectrum disorder.

Daimon, CM; Jasien, Joan M; Martin, B; Maudsley, S; Shapiro, BK; Wang, R (Front Aging Neurosci, 2014)

Autism spectrum disorder (ASD) is a complex heterogeneous neurodevelopmental disorder characterized by alterations in social functioning, communicative abilities, and engagement in repetitive or restrictive behaviors. The ...