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    Multiple endocytic pathways of G protein-coupled receptors delineated by GIT1 sensitivity. 

    Achiriloaie, M; Albanesi, JP; Claing, A; Lefkowitz, Robert J; Perry, SJ; Premont, Richard Thomas; Walker, JK (Proc Natl Acad Sci U S A, 2000-02-01)
    Recently, we identified a GTPase-activating protein for the ADP ribosylation factor family of small GTP-binding proteins that we call GIT1. This protein initially was identified as an interacting partner for the G protein-coupled ...
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    Role of endocytosis in the activation of the extracellular signal-regulated kinase cascade by sequestering and nonsequestering G protein-coupled receptors. 

    Daaka, Y; Lefkowitz, Robert J; Luttrell, LM; Maudsley, S; Pierce, KL (Proc Natl Acad Sci U S A, 2000-02-15)
    Acting through a number of distinct pathways, many G protein-coupled receptors (GPCRs) activate the extracellular signal-regulated kinase (ERK)/mitogen-activated protein kinase (MAPK) cascade. Recently, it has been shown ...
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    Hybrid transgenic mice reveal in vivo specificity of G protein-coupled receptor kinases in the heart. 

    Benovic, JL; Duncan, SJ; Eckhart, AD; Koch, Walter J; Lefkowitz, Robert J; Penn, RB (Circ Res, 2000-01-07)
    G protein-coupled receptor kinases (GRKs) phosphorylate activated G protein-coupled receptors, including alpha(1B)-adrenergic receptors (ARs), resulting in desensitization. In vivo analysis of GRK substrate selectivity has ...
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    Platelet-derived growth factor receptor association with Na(+)/H(+) exchanger regulatory factor potentiates receptor activity. 

    Blitzer, JT; Hall, RA; Lefkowitz, Robert J; Luttrell, LM; Maudsley, S; Rahman, Nadeem U; Zamah, AM (Mol Cell Biol, 2000-11)
    Platelet-derived growth factor (PDGF) is a potent mitogen for many cell types. The PDGF receptor (PDGFR) is a receptor tyrosine kinase that mediates the mitogenic effects of PDGF by binding to and/or phosphorylating a variety ...
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    Preservation of myocardial beta-adrenergic receptor signaling delays the development of heart failure after myocardial infarction. 

    Glower, Donald D Jr; Hata, Jonathan Andrew; Koch, Walter J; Lefkowitz, Robert J; Shah, AS; White, DC (Proc Natl Acad Sci U S A, 2000-05-09)
    When the heart fails, there is often a constellation of biochemical alterations of the beta-adrenergic receptor (betaAR) signaling system, leading to the loss of cardiac inotropic reserve. betaAR down-regulation and functional ...
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    Catecholamines, cardiac beta-adrenergic receptors, and heart failure. 

    Koch, Walter J; Lefkowitz, Robert J; Rockman, Howard A (Circulation, 2000-04-11)
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    Wee1-regulated apoptosis mediated by the crk adaptor protein in Xenopus egg extracts. 

    Evans, EK; Kornbluth, S; Moseley, Martin Arthur III; Moyer, MB; Murakami, M; Smith, JJ; Vande Woude, G (J Cell Biol, 2000-12-25)
    Many of the biochemical reactions of apoptotic cell death, including mitochondrial cytochrome c release and caspase activation, can be reconstituted in cell-free extracts derived from Xenopus eggs. In addition, because caspase ...
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    Intracoronary adenovirus-mediated delivery and overexpression of the beta(2)-adrenergic receptor in the heart : prospects for molecular ventricular assistance. 

    Glower, Donald D Jr; Hata, Jonathan Andrew; Koch, Walter J; Kypson, Alan P; Lefkowitz, Robert J; Lilly, R Eric; Pippen, Anne; ... (10 authors) (Circulation, 2000-02-01)
    BACKGROUND: Genetic modulation of ventricular function may offer a novel therapeutic strategy for patients with congestive heart failure. Myocardial overexpression of beta(2)-adrenergic receptors (beta(2)ARs) has been shown ...
     

     

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    AuthorLefkowitz, Robert J (7)Koch, Walter J (4)Glower, Donald D Jr (2)Hata, Jonathan Andrew (2)Luttrell, LM (2)Maudsley, S (2)Shah, AS (2)Achiriloaie, M (1)Albanesi, JP (1)Benovic, JL (1)... View MoreDate Issued
    2000 (8)
    TypeJournal article (8)SubjectAnimals (8)Humans (4)Myocardium (4)Receptors, Adrenergic, beta (4)Mitogen-Activated Protein Kinases (3)Phosphorylation (3)Adenoviridae (2)beta-Adrenergic Receptor Kinases (2)Cell Cycle Proteins (2)COS Cells (2)... View MoreAffiliation of Duke Author(s)
    Clinical Science Departments (8)
    Duke (8)Duke Cancer Institute (8)
    Institutes and Centers (8)
    Medicine (8)
    Medicine, Cardiology (8)
    School of Medicine (8)Trinity College of Arts & Sciences (8)Basic Science Departments (7)Biochemistry (7)... View More