A novel DNA damage-induced alternative splicing pathway that regulates p53 and cellular senescence markers.

dc.contributor.author	Chen, Jing
dc.contributor.author	Kastan, Michael B
dc.coverage.spatial	United States
dc.date.accessioned	2017-12-12T20:33:05Z
dc.date.available	2017-12-12T20:33:05Z
dc.date.issued	2017-09
dc.identifier	https://www.ncbi.nlm.nih.gov/pubmed/29142900
dc.identifier	367
dc.identifier.issn	2331-4737
dc.identifier.uri	https://hdl.handle.net/10161/15856
dc.language	eng
dc.publisher	Impact Journals, LLC
dc.relation.ispartof	Oncoscience
dc.relation.isversionof	10.18632/oncoscience.367
dc.subject	DNA damage
dc.subject	alternative splicing
dc.subject	p53
dc.subject	senescence
dc.title	A novel DNA damage-induced alternative splicing pathway that regulates p53 and cellular senescence markers.
dc.type	Journal article
pubs.author-url	https://www.ncbi.nlm.nih.gov/pubmed/29142900
pubs.begin-page	122
pubs.end-page	123
pubs.issue	9-10
pubs.organisational-group	Basic Science Departments
pubs.organisational-group	Clinical Science Departments
pubs.organisational-group	Duke
pubs.organisational-group	Duke Cancer Institute
pubs.organisational-group	Institutes and Centers
pubs.organisational-group	Pediatrics
pubs.organisational-group	Pharmacology & Cancer Biology
pubs.organisational-group	School of Medicine
pubs.organisational-group	Temp group - logins allowed
pubs.publication-status	Published online
pubs.volume	4

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