Abi1 loss drives prostate tumorigenesis through activation of EMT and non-canonical WNT signaling

dc.contributor.author

Nath, Disharee

dc.contributor.author

Li, Xiang

dc.contributor.author

Mondragon, Claudia

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Post, Dawn

dc.contributor.author

Chen, Ming

dc.contributor.author

White, Julie R

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Hryniewicz-Jankowska, Anita

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Caza, Tiffany

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Kuznetsov, Vladimir A

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Hehnly, Heidi

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Jamaspishvili, Tamara

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Berman, David M

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Zhang, Fan

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Kung, Sonia HY

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Fazli, Ladan

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Gleave, Martin E

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Bratslavsky, Gennady

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Pandolfi, Pier Paolo

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Kotula, Leszek

dc.date.accessioned

2019-09-20T23:40:16Z

dc.date.available

2019-09-20T23:40:16Z

dc.date.issued

2019-12

dc.date.updated

2019-09-20T23:40:04Z

dc.identifier.issn

1478-811X

dc.identifier.uri

https://hdl.handle.net/10161/19322

dc.language

en

dc.publisher

Springer Science and Business Media LLC

dc.relation.ispartof

Cell Communication and Signaling

dc.relation.isversionof

10.1186/s12964-019-0410-y

dc.title

Abi1 loss drives prostate tumorigenesis through activation of EMT and non-canonical WNT signaling

dc.type

Journal article

duke.contributor.orcid

Chen, Ming|0000-0002-3470-1062

pubs.issue

1

pubs.organisational-group

School of Medicine

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Duke

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Duke Cancer Institute

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Institutes and Centers

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Pathology

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Clinical Science Departments

pubs.publication-status

Published online

pubs.volume

17

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