Barriers and Predictors of Long-Term Physical Activity Maintenance: The STRRIDE I Reunion Cohort.
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2025-01
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Abstract
Introduction
This study aimed to identify barriers and predictors of self-reported physical activity (PA) maintenance 10 yr following the Studies of a Targeted Risk Reduction Intervention through Defined Exercise (STRRIDE I) randomized trial among young older adults.Methods
Participants completed a PA recall questionnaire that assessed barriers to PA maintenance. Chi-square tests assessed differences in barriers by PA maintenance tertile. Demographic and clinical measures collected at baseline and post-intervention from the STRRIDE I parent trial were included in model development to identify predictors of PA maintenance. Three models were generated using a backward bootstrap variable selection algorithm followed by multiple linear regression.Results
Participants who returned for the STRRIDE I Reunion study (n = 104; 63.0 ± 6.2 yr old) reported mean PA participation of 77.9 ± 76.5 min·wk-1. Commonly reported barriers to PA maintenance included lack of self-motivation (41%), time constraints (33%), illness or injury (29%), and family obligations (23%). There was a significant association between the percentage of individuals who reported one or more barriers versus no barriers by PA maintenance tertile (frequency of PA: χ 2 ratio = 26.1, P < 0.0001; amount of PA: χ 2 ratio = 15.1, P = 0.0005). The baseline predictive model had an adjusted R 2 value of 0.05, the post-intervention predictive model had an adjusted R 2 of 0.12, and the change score (post-intervention minus pre-intervention) predictive model had an adjusted R 2 of 0.17.Conclusions
Maintaining PA beyond a structured exercise intervention setting continues to be challenging for older adults. Compared to those who reported no barriers, young older adults who reported one or more barriers to PA maintenance were less active 10 yr following STRRIDE I. Additionally, how participants respond to a structured exercise intervention in certain clinical variables may be the most indicative of future PA maintenance.Type
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Collins-Bennett, Katherine A, Leanna M Ross, Johanna L Johnson, Cris A Slentz, Kim M Huffman and William E Kraus (2025). Barriers and Predictors of Long-Term Physical Activity Maintenance: The STRRIDE I Reunion Cohort. Translational journal of the American College of Sports Medicine, 10(1). p. e000276. 10.1249/tjx.0000000000000276 Retrieved from https://hdl.handle.net/10161/33849.
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Scholars@Duke
Katherine Collins-Bennett
Katherine A. Collins-Bennett, PhD, NBC-HWC, is a Medical Instructor in the Department of Population Health Sciences and affiliated with the Duke Molecular Physiology Institute at Duke University School of Medicine, and is a board-certified health and wellness coach. She studies barriers and predictors of health-promoting behavior change. The ultimate goal of her translational research is to design trials to optimize health-promoting behaviors for those at risk for "relapse" or ceased behavioral modification, in order to improve long-term health and well-being.
Leanna Ross
Dr. Ross's research focuses on understanding the mechanisms by which exercise interventions elicit short- and long-term cardiometabolic health benefits. As cardiometabolic disease remains the leading cause of morbidity and mortality in the United States, the goal of her translational research is to enhance the development of evidence-based, precision exercise interventions that optimally prevent and treat disease.
Areas of Research Interest
Exercise dose-response and cardiometabolic health
Insulin action and glucose homeostasis
Legacy health benefits of exercise
Heterogeneity of response to exercise intervention
Precision lifestyle medicine
Epidemiology of physical activity and cardiorespiratory fitness
Kim Marie Huffman
Determining the role of physical activity in modulating health outcomes (cardiovascular disease risk) in persons with rheumatologic diseases (rheumatoid arthritis, gout, osteoarthritis)
Integrating clinical rheumatology, basic immunology, metabolism, and exercise science in order to reduce morbidity in individuals with arthritis
Evaluating relationships between circulating and intra-muscular metabolic intermediates and insulin resistance in sedentary as well as individuals engaging in regular exercise
Addressing the role of physical activity in modulating inflammation, metabolism, and functional health in aging populations
William Erle Kraus
My training, expertise and research interests range from human integrative physiology and genetics to animal exercise models to cell culture models of skeletal muscle adaptation to mechanical stretch. I am trained clinically as an internist and preventive cardiologist, with particular expertise in preventive cardiology and cardiac rehabilitation. My research training spans molecular biology and cell culture, molecular genetics, and integrative human exercise physiology and metabolism. I practice as a preventive cardiologist with a focus on cardiometabolic risk and exercise physiology for older athletes. My research space has both a basic wet laboratory component and a human integrative physiology one.
One focus of our work is an integrative physiologic examination of exercise effects in human subjects in clinical studies of exercise training in normal individuals, in individuals at risk of disease (such as pre-diabetes and metabolic syndrome; STRRIDE), and in individuals with disease (such as coronary heart disease, congestive heart failure and cancer).
A second focus of my research group is exploration of genetic determinates of disease risk in human subjects. We conduct studies of early onset cardiovascular disease (GENECARD; CATHGEN), congestive heart failure (HF-ACTION), peripheral arterial disease (AMNESTI), and metabolic syndrome. We are exploring analytic models of predicting disease risk using established and innovative statistical methodology.
A third focus of my group’s work is to understand the cellular signaling mechanisms underlying the normal adaptive responses of skeletal muscle to physiologic stimuli, such as occur in exercise conditioning, and to understand the abnormal maladaptive responses that occur in response to pathophysiologic stimuli, such as occur in congestive heart failure, aging and prolonged exposure to microgravity.
Recently we have begun to investigate interactions of genes and lifestyle interventions on cardiometabolic outcomes. We have experience with clinical lifestyle intervention studies, particularly the contributions of genetic variants to interventions responses. We call this Lifestyle Medicopharmacogenetics.
KEY WORDS:
exercise, skeletal muscle, energy metabolism, cell signaling, gene expression, cell stretch, heart failure, aging, spaceflight, human genetics, early onset cardiovascular disease, lifestyle medicine
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