Abi1 loss drives prostate tumorigenesis through activation of EMT and non-canonical WNT signaling
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https://hdl.handle.net/10161/19322Published Version (Please cite this version)
10.1186/s12964-019-0410-yPublication Info
Nath, Disharee; Li, Xiang; Mondragon, Claudia; Post, Dawn; Chen, Ming; White, Julie
R; ... Kotula, Leszek (2019). Abi1 loss drives prostate tumorigenesis through activation of EMT and non-canonical
WNT signaling. Cell Communication and Signaling, 17(1). 10.1186/s12964-019-0410-y. Retrieved from https://hdl.handle.net/10161/19322.This is constructed from limited available data and may be imprecise. To cite this
article, please review & use the official citation provided by the journal.
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Show full item recordScholars@Duke
Ming Chen
Associate Professor in Pathology
Our laboratory is interested in understanding the molecular and genetic events underlying
cancer progression and metastasis. The focus of our work is a series of genetically
engineered mouse models that faithfully recapitulate human disease. Using a combination
of mouse genetics, omics technologies, cross-species analyses and in vitro approaches,
we aim to identify cancer cell–intrinsic and –extrinsic mechanisms driving
metastatic cancer progression, with a long

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