A peptide of the N terminus of GRK5 attenuates pressure-overload hypertrophy and heart failure

Abstract

<jats:p>GRK5 does not mediate pathological signaling in the heart if its nuclear translocation is disrupted.</jats:p>

Department

Description

Provenance

Subjects

Citation

Published Version (Please cite this version)

10.1126/scisignal.abb5968

Publication Info

Coleman, Ryan C, Akito Eguchi, Melissa Lieu, Rajika Roy, Eric W Barr, Jessica Ibetti, Anna-Maria Lucchese, Amanda M Peluzzo, et al. (2021). A peptide of the N terminus of GRK5 attenuates pressure-overload hypertrophy and heart failure. Science Signaling, 14(676). 10.1126/scisignal.abb5968 Retrieved from https://hdl.handle.net/10161/28930.

This is constructed from limited available data and may be imprecise. To cite this article, please review & use the official citation provided by the journal.

Scholars@Duke

Roy

Rajika Roy

Assistant Professor in Surgery

Unless otherwise indicated, scholarly articles published by Duke faculty members are made available here with a CC-BY-NC (Creative Commons Attribution Non-Commercial) license, as enabled by the Duke Open Access Policy. If you wish to use the materials in ways not already permitted under CC-BY-NC, please consult the copyright owner. Other materials are made available here through the author’s grant of a non-exclusive license to make their work openly accessible.