A peptide of the N terminus of GRK5 attenuates pressure-overload hypertrophy and heart failure

dc.contributor.author

Coleman, Ryan C

dc.contributor.author

Eguchi, Akito

dc.contributor.author

Lieu, Melissa

dc.contributor.author

Roy, Rajika

dc.contributor.author

Barr, Eric W

dc.contributor.author

Ibetti, Jessica

dc.contributor.author

Lucchese, Anna-Maria

dc.contributor.author

Peluzzo, Amanda M

dc.contributor.author

Gresham, Kenneth

dc.contributor.author

Chuprun, J Kurt

dc.contributor.author

Koch, Walter J

dc.date.accessioned

2023-09-01T16:12:41Z

dc.date.available

2023-09-01T16:12:41Z

dc.date.issued

2021-03-30

dc.date.updated

2023-09-01T16:12:40Z

dc.description.abstract

<jats:p>GRK5 does not mediate pathological signaling in the heart if its nuclear translocation is disrupted.</jats:p>

dc.identifier.issn

1945-0877

dc.identifier.issn

1937-9145

dc.identifier.uri

https://hdl.handle.net/10161/28930

dc.language

en

dc.publisher

American Association for the Advancement of Science (AAAS)

dc.relation.ispartof

Science Signaling

dc.relation.isversionof

10.1126/scisignal.abb5968

dc.title

A peptide of the N terminus of GRK5 attenuates pressure-overload hypertrophy and heart failure

dc.type

Journal article

duke.contributor.orcid

Roy, Rajika|0000-0001-9394-3224

duke.contributor.orcid

Koch, Walter J|0000-0002-8522-530X

pubs.issue

676

pubs.organisational-group

Duke

pubs.organisational-group

School of Medicine

pubs.organisational-group

Clinical Science Departments

pubs.organisational-group

Surgery

pubs.organisational-group

Surgery, Cardiovascular and Thoracic Surgery

pubs.publication-status

Published

pubs.volume

14

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